Education
| School — Major — Degree | Duration |
|---|---|
|
國立臺北科技大學 — 工程科技研究所生化與生醫工程組(畢) — 博士 Doctorate, Department of chemical engineering and biotechnology, National Taipei University of Technology |
2008/09 — 2013/06 |
|
東海大學 — 生物學系(畢) — 碩士 Master, Department of biology, TungHai University |
1999/09 — 2001/06 |
|
東海大學 — 生物學系(畢) — 學士 Bachelor, Department of biology, TungHai University |
1995/09 — 1999/06 |
Intramural Experience
| Office/Department/Institute | Position | Duration |
|---|---|---|
| 生物醫學暨環境生物學系 Department of Biomedical Science and Environmental Biology | Assistant Professor | 2023/08/01 — |
| 生物醫學暨環境生物學系 Department of Biomedical Science and Environmental Biology | Assistant Professor | 2021/08/01 — 2023/07/31 |
Extramural Experience
| DEPNO | Position- Office/Department/Institute | Duration |
|---|---|---|
| 日本京都大學 | 特定研究員 | 1041116 — 1100630 |
Discipline
Research & Technology Platform Open to the Outside
Interested Area(s)for Interdisciplinary Research
Area(s) of Expertise & Research
幹細胞生物學、生醫材料與組織工程
Stem cell biology, Biomaterials and Tissue engineering
Publication
| NO | Publication |
|---|---|
| 1 | 人類乳牙幹細胞的外泌體可抑制骨關節炎的發炎 The Exosomes of Stem Cells from Human Exfoliated Deciduous Teeth Suppress Inflammation in Osteoarthritis. |
| 2 | ETFDH 基因突變透過 Bcl2 訊息造成過度細胞凋亡和神經突起生長缺陷 ETFDH mutation involves excessive apoptosis and neurite outgrowth defect via Bcl2 pathway. |
| 3 | SMN promotes mitochondrial metabolic maturation during myogenesis by, regulating the MYOD-miRNA axis SMN promotes mitochondrial metabolic maturation during myogenesis by, regulating the MYOD-miRNA axis |
| 4 | Effects of Organic Elicitors on the Recycled Production of Ginkgolide B in Immobilized Cell Cultures of Ginkgo biloba Effects of Organic Elicitors on the Recycled Production of Ginkgolide B in Immobilized Cell Cultures of Ginkgo biloba |
| 5 | Healing of a bone-exposed soft tissue defect after surgical correction of traumatic patellar dislocation: A case report. Healing of a bone-exposed soft tissue defect after surgical correction of traumatic patellar dislocation: A case report. |
| 6 | The Potential Application and Promising Role of Targeted Therapy in, Pulmonary Arterial Hypertension The Potential Application and Promising Role of Targeted Therapy in, Pulmonary Arterial Hypertension |
| 7 | 幹細胞療法於糖尿病傷口之應用 Stem Cell-Based Therapeutic Strategies in Diabetic Wound Healing. |
| 8 | Opportunities and Challenges of Human IPSC Technology in Kidney Disease Research Opportunities and Challenges of Human IPSC Technology in Kidney Disease Research |
| 9 | 肺門脈高壓臨床前標靶治療之探討 The Potential Application and Promising Role of Targeted Therapy in Pulmonary Arterial Hypertension |
| 10 | Micronized sacchachitin promotes satellite cell proliferation through, TAK1-JNK-AP-1 signaling pathway predominantly by TLR2 activation (vol, 15, 100, 2020) Micronized sacchachitin promotes satellite cell proliferation through, TAK1-JNK-AP-1 signaling pathway predominantly by TLR2 activation (vol, 15, 100, 2020) |
| 11 | Bidirectional myofiber transition through altering the, photobiomodulation condition Bidirectional myofiber transition through altering the, photobiomodulation condition |
| 12 | Muscle type from which satellite cells are derived plays a role in their, damage response Muscle type from which satellite cells are derived plays a role in their, damage response |
| 13 | In vitro Neuromuscular Junction Induced from Human Induced Pluripotent, Stem Cells In vitro Neuromuscular Junction Induced from Human Induced Pluripotent, Stem Cells |
| 14 | Micronized sacchachitin promotes satellite cell proliferation through, TAK1-JNK-AP-1 signaling pathway predominantly by TLR2 activation Micronized sacchachitin promotes satellite cell proliferation through, TAK1-JNK-AP-1 signaling pathway predominantly by TLR2 activation |
| 15 | A transient protective effect of low-level laser irradiation against, disuse-induced atrophy of rats A transient protective effect of low-level laser irradiation against, disuse-induced atrophy of rats |
| 16 | iPSC-derived functional human neuromuscular junctions model the, pathophysiology of neuromuscular diseases iPSC-derived functional human neuromuscular junctions model the, pathophysiology of neuromuscular diseases |
| 17 | Adaptable interaction between aquaporin-1 and band 3 reveals a potential, role of water channel in blood CO2 transport Adaptable interaction between aquaporin-1 and band 3 reveals a potential, role of water channel in blood CO2 transport |
| 18 | Interaction of glycosphingolipids GD3 and GD2 with growth factor, receptors maintains breast cancer stem cell phenotype Interaction of glycosphingolipids GD3 and GD2 with growth factor, receptors maintains breast cancer stem cell phenotype |
| 19 | Study of subcellular dynamics on cell-substrate interactions by live, cell imaging Study of subcellular dynamics on cell-substrate interactions by live, cell imaging |
| 20 | Three dimensional chitosan scaffolds influence the extra cellular matrix, expression in Schwann cells Three dimensional chitosan scaffolds influence the extra cellular matrix, expression in Schwann cells |
| 21 | Heat-Induced Antigen Retrieval Applied in Zebrafish: Whole-Mount In Situ, Immunofluorescence Microscopy Heat-Induced Antigen Retrieval Applied in Zebrafish: Whole-Mount In Situ, Immunofluorescence Microscopy |
| 22 | Increased cellular apoptosis susceptibility (CSE1L/CAS) protein, expression promotes protrusion extension and enhances migration of MCF-7, breast cancer cells Increased cellular apoptosis susceptibility (CSE1L/CAS) protein, expression promotes protrusion extension and enhances migration of MCF-7, breast cancer cells |
| 23 | Micropillar substrate influences the cellular attachment and laminin expression. Micropillar substrate influences the cellular attachment and laminin expression. |
| 24 | CSEIL/CAS, the cellular apoptosis susceptibility protein, enhances, invasion and metastasis but not proliferation of cancer cells CSEIL/CAS, the cellular apoptosis susceptibility protein, enhances, invasion and metastasis but not proliferation of cancer cells |
Project
| NO |
YEAR — Source — No — Type
Project Name
|
Duration |
|---|---|---|
| 1 |
115 — N 國科會(原科技部) — NSTC113-2314-B-037-082-MY3 — 1 個人型
以人類多功能幹細胞建構之功能性神經肌肉接頭模擬先天性肌無力綜合症以探討運動神經元與骨骼肌之間的交互作用與病理發展(3/3)( )
|
2026/08/01 ~ 2027/07/31 |
| 2 |
114 — N 國科會(原科技部) — NSTC113-2314-B-037-082-MY3 — 1 個人型
以人類多功能幹細胞建構之功能性神經肌肉接頭模擬先天性肌無力綜合症以探討運動神經元與骨骼肌之間的交互作用與病理發展(2/3)( )
|
2025/08/01 ~ 2027/07/31 |
| 3 |
113 — N 國科會(原科技部) — NSTC113-2314-B-037-082-MY3 — 1 個人型
以人類多功能幹細胞建構之功能性神經肌肉接頭模擬先天性肌無力綜合症以探討運動神經元與骨骼肌之間的交互作用與病理發展(1/3)( )
|
2024/08/01 ~ 2027/07/31 |
| 4 |
113 — NK 高醫-中山合作計畫 — NK113P28 — 1 個人型
應用新型微矩陣細胞篩選技術建構人類iPS細胞產生的體外疾病模型—以神經肌肉接頭疾病模式為例(Application of a novel microarray cell-sorting technique to generate an in vitro disease model by human iPScells — An example of neuromuscular junction disease model)
|
2024/01/01 ~ 2024/12/31 |
| 5 |
112 — N 國科會(原科技部) — NSTC112-2314-B-037-077 — 1 個人型
人類多功能幹細胞建構之功能性神經肌肉接頭用於模擬相關疾病—以先天性肌無力綜合症為例( )
|
2023/08/01 ~ 2024/07/31 |
| 6 |
112 — NK 高醫-中山合作計畫 — — 1 個人型
應用FRET分子力顯微鏡研究以人類iPSC誘導分化之神經肌肉接頭模擬之先天性綜合肌肉萎縮症 (CMS)的功能變異(Study of functional alternations of congenital myasthenic syndrome using human iPSC derived neuromuscular junction by FRET-based molecular force microscopy)
|
2023/01/01 ~ 2023/12/31 |
| 7 |
112 — Z 大專學生研究計畫及博士後 — NSTC113-2813-C-037-038-B — 8 大專學生研究計畫
113年度大專學生研究計畫-徐艾(生物3)( )
|
2024/07/01 ~ 2025/02/28 |
| 8 |
111 — DK 前瞻重點(原校院) — KMU-DK(B)112001-1 — 2 整合型
設計合成藉葡萄糖醛 酸轉換化性之抗癌藥 物(monomethylauristatin E-G, MMAE-G)以增加微脂 體傳遞藥物應用於癌 症治療( )
|
2023/01/01 ~ 2023/12/31 |
| 9 |
111 — N 國科會(原科技部) — MOST111-2314-B-037-060 — 1 個人型
以人類iPS細胞的疾病模式研究先天性肌無力綜合症之基因變異( )
|
2022/08/01 ~ 2023/07/31 |
| 10 |
111 — Q 新聘教師計畫(校內) — KMU-Q112003 —
利用誘導性多功能幹細胞建構人類先天性肌無力綜合症之疾病模式( )
|
2023/01/01 ~ 2023/12/31 |
| 11 |
111 — Z 大專學生研究計畫及博士後 — NSTC112-2813-C-037-039-B — 8 大專學生研究計畫
112年度大專學生研究計畫-陳佳珮(生物3)( )
|
2023/07/01 ~ 2024/02/29 |
| 12 |
111 — Z 大專學生研究計畫及博士後 — NSTC112-2813-C-037-040-B — 8 大專學生研究計畫
112年度大專學生研究計畫-凃宇音(生物3)( )
|
2023/07/01 ~ 2024/02/29 |
| 13 |
110 — Q 新聘教師計畫(校內) — KMU-Q111005 —
人類iPS細胞建構的肌肉-神經組織與生醫材料之間的交互作用( )
|
2022/01/01 ~ 2022/12/31 |